A widely-used herbicide in the US has turned frogs gay, so what is it doing to humans?

By Mike Adams of Natural News

As the lab science director of CWC Labs, I not only conduct mass spec analyses of pesticides and herbicides in foods, but I also have access to a massive library of published books describing the toxic effects of hundreds of different pesticides. Atrazine is widely documented as a “powerful chemical castrator” that transforms males into hermaphrodites, the animal kingdom version of a “metrosexual.” It’s also widely present in the U.S. water supply. It is undoubtedly one of the chemicals currently responsible for the mass feminization of men in modern society.

To explain the science, I’m republishing an excerpt from the book Our Daily Poison – From Pesticides to Packaging How Chemicals Have Contaminated the Food Chain and Are Making Us Sick by Marie-Monique Robin, published in 2014.

“The males became homosexuals and coupled with other males, adopting a feminized behavior… atrazine acted as a very powerful chemical castrator”

The most astonishing and relevant passage from this book is as follows, relating the findings from a brilliant scientist (Tyrone Hayes) who studied the effects of atrazine on males and the endocrine system:

“We observed that atrazine reduced the size of the larynx, which is the voice box in the males. Since they sing to seduce the females, this meant they were sexually handicapped. We also observed very low levels of testosterone among the adult males; some of them were hermaphrodites, which means they had both ovaries and testes. In certain cases, the males became homosexuals and coupled with other males, adopting a feminized behavior; sometimes they had eggs in their testes instead of sperm. Ultimately, atrazine acted as a very powerful chemical castrator that is biologically active at 1 ppb, and even 0.1 ppb.”

Atrazine: A “Powerful Chemical Castrator”

Excerpted from the book, bolding added for emphasis

During the New Orleans symposium, Tyrone Hayes evoked one of his latest studies showing that atrazine, an agricultural poison, provoked mechanisms characteristic of breast and prostate cancers in human cells exposed to doses similar to those found in the environment.*

“You’ve all heard the good news,” he exclaimed. “The Environmental Protection Agency [EPA] has announced that it will reexamine atrazine’s scientific file! Let’s hope it will end up banning it just like Europe did five years ago!”

Though the herbicide was banned by the European Union in 2004, it is still massively used throughout the United States, where approximately forty thousand tons are spread on countless farms growing crops such as corn, sorghum, sugar cane, and wheat every year.’’

Lauded as the “DDT for weeds” when it was put on the market in 1958, today atrazine is the principal contaminant of American surface and ground waters, much like in the majority of European countries (with France in the lead), despite the ban.

Two weeks before the New Orleans symposium, Lisa Jackson, the EPA director nominated by President Barack Obama in January 2009, had effectively announced that the agency would “conduct a new evaluation of the pesticide to assess any possible links between atrazine and cancer, as well as other health problems, such as premature births.”

“This is a dramatic change,” said Linda Birnbaum, director of the National Institute of Environmental Health Sciences (NIEHS) (see Chapter 18). “There is growing evidence that atrazine could be a hazard to human health. This is a strong signal that the world is changing in regards to some of the most widely used chemicals.”

If there is one scientist who battled for atrazine’s ban in the United States, it is inarguably Tyrone Hayes, even if (as he explained to me during our meeting in his Berkeley laboratory on December 12, 2009) “this battle wasn’t a personal decision, but was imposed by events.”

In 1998, he was contacted by Novartis (the company became Syngenta two years later after its merger with AstraZeneca), which offered him a “handsomely paid” contract to “verify if atrazine [was] an endocrine disruptor,” as Theo Colborn and her co-authors note in Our Stolen Future (see Chapter 16).

For the industry, the matter was quite serious as, seven years earlier, a U.S. Geological Survey report had revealed that “atrazine exceeded drinking water standards in 27 percent of the samples” taken from the Missouri, Mississippi, and Ohio Bivers and tributaries. What’s more, in the 1980s, two studies conducted on mice and rats had indicated that exposure to the herbicide brought on breast and uterine cancers, lymphomas, and leukemia.

The International Agency for Research on Cancer (IARC) judged the results sufficiently convincing and decided to classify atrazine as “possibly carcinogenic to humans” (group 2B) in 1991. As a result, the EPA, leaning on the Safe Drinking Water Act, decreased the atrazine standard to a maximum of 3 ug/l of water, or 3 ppb (parts per billion). In 1994, three studies established a link between rodents’ exposure to atrazine and mammary tumors.

Then in 1997,  an epidemiological study carried out in several rural Kentucky counties found a significant excess of breast cancer among the most exposed women (in correlation with the level of water contamination and proximity of the home to corn cultivations).

Thus began Novartis’ great strategic era. Its first tactic was tremendously effective, and brought about IARC’s downgrade of atrazine from group 2B to group 3 (not classifiable) in 1999. To justify the surprising decision, the UN agency experts relied on a line of reasoning I described in Chapter 10: “the mechanism by which atrazine increases the incidence of mammary gland tumors in rats is not relevant to humans.”

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Thanks to Mike Adams of Natural News for this article, the rest of which you can read here.

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